fig10

Figure 10. (A and B) TEM images of g-C₃N₄/PDINH heterostructures. (C) Photographs of the infected wound treated in different days and the infected wound histologic analyses in day 1 and day 6 with different treatments. The scale bar in the photographs is 0.5 cm and, in the histologic picture, is 100 μm. (Reproduced with permission^[109]. Copyright 2019, Wiley-VCH). (D) Proposed pCN dissolution mechanism. (E) Photographs of the as-prepared free-standing pCN/CNT membrane, 180° folded profile, and tailored ones of different shapes. (F) Scheme of pCN and CNTs in the composite membrane. (G) Energy band position of g-C₃N₄ on the pCN/CNT membrane with respect to ROS formation potential. (H) Photographs of S. aureus-infected wounds in laboratory mice treated with the pCN/CNT membrane and control gauze at different times. (I) Histologic analysis (HE, Masson, CD31) of the S. aureus-infected wounds after treatment with the pCN/CNT membrane and control gauze for nine days. (Reproduced with permission^[110]. Copyright 2023, Royal Society of Chemistry). (J) Scheme illustrated the synthesis of g-C₃N₄/ADN and corresponding bacterial killing and biofilm elimination activities. Eradication of mature S. mutans biofilm by the synthesized NSs. (K) Pictures of 48 h mature S. mutans biofilm on tooth models incubated with PBS, g-C₃N₄, g-C₃N₄/ADN, g-C₃N₄ + light, and g-C₃N₄/ADN + light. (L) Remaining biofilm biomass determined by CV staining after 3 h of incubation. (M) Scheme illustrates the antibacterial mechanism of g-C₃N₄/ADN. (Reproduced with permission^[111]. Copyright 2022, Elsevier).